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However, testosterone in physiologic doses could even be beneficial in sufferers with angina , have higher ischemic thresholds and improved quality of life . Short-term intracoronary administration of testosterone induces coronary artery dilation and will increase coronary blood flow in men with established coronary artery disease . Therefore, an interaction of endogenous elements of the patient’s threat profile as well as the testosterone dosing could additionally be problems with whether antagonistic occasions occur. Erythrocytosis has been reported as a potential impact of testosterone, which may be beneficial in sufferers with anemia.

Patients additionally could also be taking concomitant stimulants, corresponding to ephedra, which might confound information and the potential for adverse events. It is most likely going that the frequency of cardiac occasions is underreported within the medical literature. Moreover, testosterone has been used within the remedy of reduced muscle mass and bone density in addition to anemia. The role testosterone might play in the growth of cardiovascular disease has increasingly become the subject of current interest . Supraphysiologic doses of anabolic-androgenic steroids , which include greater than 30 natural and synthetic derivatives of testosterone, may cause a wide spectrum of clinical sequalae. Supplemental or physiologic doses of testosterone have noticed expanded use in medical practice just lately. Other common potential antagonistic results embrace infertility, hepatotoxicity, psychological abnormalities, and gynecomastia .

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We report a case of an acute ST-segment elevation myocardial infarction in a patient with traditional cardiac risk components using supraphysiologic doses of supplemental, intramuscular testosterone. In addition, this affected person additionally had polycythemia, probably secondary to high-dose testosterone. The affected person underwent profitable percutaneous intervention of the best coronary artery. Physicians and sufferers ought to be conscious of the clinical consequences of anabolic steroid abuse. Development of an atheromatous plaque perpetuates endothelial dysfunction and promotes platelet aggregation and intracoronary thrombus formation (Ajayi et al 1995; Nieminen et al 1996). Moreover, a component of endothelial dysfunction has been proposed, which may contribute to irregular vessel reactivity.